Cannabis oil for cancer treatments is provided by CBD International. Our treatment has helped thousands of cancer patients with their condition!. I don't spout CBD oil as a cancer cure I don't know enough about it yet to spout. My workmate showed evidence that her mum in laws 10 liver. Whole or crude marijuana (including marijuana oil or hemp oil) is not approved by the US Food and Drug Administration (FDA) for any medical.
& oil Cancer CBD
I still don't regret it though. Its had a positive effect, no bad side effects and Ive felt like I have done somthing. Just like to mention I am a Social Scientist by trade so I seek scientific research. Nothing less will do. I will have a look at the links you have kindly provided.
I would never risk giving the most precious thing in my life anything I thought would hasten him away from me. Indeed even when I made the choice of product, I discussed it with him in detail and first tried it on myself.
I am really grateful for all viewpoints and assistance. I read on here someone refused chemo in favour of canabis oil. That is a brave step. I personally would not turn my back on conventional treatments completely, but equally don't condemn that. Cancer is rife in hubbys family; one sister chose a very different path to us. After diagnosis, she chose to give up. No food, no meds, and less than a month later we went to the funeral.
I saw benefits as well as downers to her decision; yes she missed months if not a year or so with the family, very sad, but also saved herself and them a lot of suffering,worry and fear. The British Medical journal has some interesting articles meriting further attention. Some sillier than others, but a bit more palatable.
The main thing is making the effort to look. No one should be dismissed because whatever they read wasn't science based enough. It is important to listen to others' experiences. That includes listening to people on here. If someone's dad had a cancer that went away after treatemnt only with custard. My attitude isn't " not scientific, go away", it's "Really, then lets look at this right away! This forum reaffirmed my own experience about how disconnected many carers are from the world out there.
What a wealth of people there are who desperately want to 'offer support' maybe especially 'professionals', but when the chips are down, it is a very lonely path, not for everyone, but those who can't fit the 9 to 5, those living remotely, those who can't drive nor live on the hospice doorstep When I realised this for the first time despite an honours degree in the science of society, my eyes were like dinner plates!
Academia was once a drug to me but without commonsense and worldly experience Real experiences people on here share are what have upped my game so far. Here here well said So thank you for starting this thread Delta 9 -tetrahydrocannabinol inhibits 17beta-estradiol-induced proliferation and fails to activate androgen and estrogen receptors in MCF7 human breast cancer cells.
JunD is involved in the antiproliferative effect of Delta9-tetrahydrocannabinol on human breast cancer cells. Anandamide inhibits Cdk2 and activates Chk1 leading to cell cycle arrest in human breast cancer cells. Toxicological profiles of selected synthetic cannabinoids showing high binding affinities to the cannabinoid receptor subtype CB 1 Arch Toxicol.
Cannabidiol induces programmed cell death in breast cancer cells by coordinating the cross-talk between apoptosis and autophagy. A high cannabinoid CB 1 receptor immunoreactivity is associated with disease severity and outcome in prostate cancer.
Increased expressions of cannabinoid receptor-1 and transient receptor potential vanilloid-1 in human prostate carcinoma. J Cancer Res Clin Oncol.
Delta9-tetrahydrocannabinol induces apoptosis in human prostate PC-3 cells via a receptor-independent mechanism. Involvement in Raf-1 stimulation and NGF induction. Cannabinoid receptor as a novel target for the treatment of prostate cancer. Cannabinoid receptor-dependent and -independent anti-proliferative effects of omega-3 ethanolamides in androgen receptor-positive and -negative prostate cancer cell lines.
Guindon J, Hohmann AG. The endocannabinoid system and cancer: The putative cannabinoid receptor GPR55 defines a novel autocrine loop in cancer cell proliferation. Involvement of CB1 cannabinoid receptor and Raf Anti-proliferative and apoptotic effects of anandamide in human prostatic cancer cell lines: Induction of apoptosis by cannabinoids in prostate and colon cancer cells is phosphatase dependent.
Molecular characterization of an enzyme that degrades neuromodulatory fatty-acid amides. Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase.
Anti-proliferative and anti-angiogenic effects of CB2R agonist JWH in non-small lung cancer cells A and human umbilical vein endothelial cells: Folia Biol Praha ; 58 2: Cannabinoid receptors as novel targets for the treatment of melanoma. Cannabinoids in pancreatic cancer: Cannabinoids induce apoptosis of pancreatic tumor cells via endoplasmic reticulum stress-related genes. Cannabinoid derivatives induce cell death in pancreatic MIA PaCa-2 cells via a receptor-independent mechanism.
Cartilage tumours and bone development: Management of bone metastases. A decrease in anandamide signaling contributes to the maintenance of cutaneous mechanical hyperalgesia in a model of bone cancer pain. Multicenter, double-blind, randomized, placebo-controlled, parallel-group study of the efficacy, safety, and tolerability of THC: J Pain Symptom Manage.
Differential effects of repeated low dose treatment with the cannabinoid agonist WIN 55, in experimental models of bone cancer pain and neuropathic pain. A cannabinoid 2 receptor agonist attenuates bone cancer-induced pain and bone loss. The cannabinoid receptor agonist, WIN 55, , attenuates tumor-evoked hyperalgesia through peripheral mechanisms. Acute and chronic administration of the cannabinoid receptor agonist CP 55, attenuates tumor-evoked hyperalgesia.
Reduction of bone cancer pain by activation of spinal cannabinoid receptor 1 and its expression in the superficial dorsal horn of the spinal cord in a murine model of bone cancer pain. Spinal and peripheral analgesic effects of the CB2 cannabinoid receptor agonist AM in two models of bone cancer-induced pain.
Intrathecal administration of the cannabinoid 2 receptor agonist JWH can attenuate cancer pain and decrease mRNA expression of the 2B subunit of N-methyl-D-aspartic acid. Disease modification of breast cancer-induced bone remodeling by cannabinoid 2 receptor agonists.
J Bone Miner Res. Inhibition of tumor angiogenesis by cannabinoids. The stress-regulated protein p8 mediates cannabinoid-induced apoptosis of tumor cells. Cannabinoids inhibit glioma cell invasion by down-regulating matrix metalloproteinase-2 expression. Cannabinoid action induces autophagy-mediated cell death through stimulation of ER stress in human glioma cells.
Triggering of the TRPV2 channel by cannabidiol sensitizes glioblastoma cells to cytotoxic chemotherapeutic agents. Cannabidiol enhances the inhibitory effects of delta9-tetrahydrocannabinol on human glioblastoma cell proliferation and survival.
A combined preclinical therapy of cannabinoids and temozolomide against glioma. Amphiregulin is a factor for resistance of glioma cells to cannabinoid-induced apoptosis. Stimulation of ALK by the growth factor midkine renders glioma cells resistant to autophagy-mediated cell death. Local delivery of cannabinoid-loaded microparticles inhibits tumor growth in a murine xenograft model of glioblastoma multiforme. Cannabinoid receptor ligands mediate growth inhibition and cell death in mantle cell lymphoma.
The role of cannabinoid receptors and the endocannabinoid system in mantle cell lymphoma and other non-Hodgkin lymphomas. Enhancing the in vitro cytotoxic activity of Delta9-tetrahydrocannabinol in leukemic cells through a combinatorial approach.
Potentiation of cannabinoid-induced cytotoxicity in mantle cell lymphoma through modulation of ceramide metabolism. Concomitant consumption of marijuana, alcohol and tobacco in oral squamous cell carcinoma development and progression: A population-based case-control study of marijuana use and head and neck squamous cell carcinoma.
Cancer Prev Res Phila ; 2 8: Cannabinoid 2 receptor induction by IL and its potential as a therapeutic target for the treatment of anaplastic thyroid carcinoma. A metabolically stable analogue of anandamide, Met-F-AEA, inhibits human thyroid carcinoma cell lines by activation of apoptosis. Cell migration in tumors. Curr Opin Cell Biol. Tumor cell-mediated neovascularization and lymphangiogenesis contrive tumor progression and cancer metastasis.
The cytoskeleton and cancer. Novel hexahydrocannabinol analogs as potential anti-cancer agents inhibit cell proliferation and tumor angiogenesis. Cannabidiolic acid, a major cannabinoid in fiber-type cannabis, is an inhibitor of MDA-MB breast cancer cell migration. Nevalainen T, Irving AJ. GPR55, a lysophosphatidylinositol receptor with cannabinoid sensitivity? Curr Top Med Chem.
Insights from transgenic mouse models of ERBB2-induced breast cancer. Cannabidiol as potential anticancer drug. Br J Clin Pharmacol. Ramer R, Hinz B. Inhibition of cancer cell invasion by cannabinoids via increased expression of tissue inhibitor of matrix metalloproteinases J Natl Cancer Inst. Cannabidiol inhibits lung cancer cell invasion and metastasis via intercellular adhesion molecule The non-psychoactive cannabidiol triggers caspase activation and oxidative stress in human glioma cells.
Cell Mol Life Sci. Antitumorigenic effects of cannabinoids beyond apoptosis. Id-1 is a key transcriptional regulator of glioblastoma aggressiveness and a novel therapeutic target.
Impact of cyclooxygenase-2 in breast cancer. Role of cannabinoid and vanilloid receptors in invasion of human breast carcinoma cells. J Environ Pathol Toxicol Oncol. Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells.
Pathways mediating the effects of cannabidiol on the reduction of breast cancer cell proliferation, invasion, and metastasis. Breast Cancer Res Treat. Cannabinoid receptors, CB1 and CB2, as novel targets for inhibition of non-small cell lung cancer growth and metastasis.
Cancer Prev Res Phila ; 4 1: Delta9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo.
Formation and regulation of the cancer stem cell niche. Effects of cannabinoids and related fatty acids upon the viability of P19 embryonal carcinoma cells.
Cannabinoids induce glioma stem-like cell differentiation and inhibit gliomagenesis. Cannabinoids inhibit energetic metabolism and induce AMPK-dependent autophagy in pancreatic cancer cells. Cannabinoids inhibit cellular respiration of human oral cancer cells. Immune regulation of cancer. Anti-tumoral action of cannabinoids: Cannabinoids inhibit peptidoglycan-induced phosphorylation of NF-kappaB and cell growth in U87MG human malignant glioma cells. Anandamide suppresses proliferation and cytokine release from primary human T-lymphocytes mainly via CB2 receptors.
Role of nuclear factor of activated T-cells and activator protein-1 in the inhibition of interleukin-2 gene transcription by cannabinol in EL4 T-cells. Anandamide is an endogenous inhibitor for the migration of tumor cells and T lymphocytes. Retrograde signalling by endocannabinoids. Cannabinoids inhibit the respiration of human sperm. Receptor mechanism and antiemetic activity of structurally-diverse cannabinoids against radiation-induced emesis in the least shrew.
Very low doses of delta 8-THC increase food consumption and alter neurotransmitter levels following weight loss. Cannabidiol enhances xenobiotic permeability through the human placental barrier by direct inhibition of breast cancer resistance protein: Am J Obstet Gynecol. Cannabidiol attenuates catalepsy induced by distinct pharmacological mechanisms via 5-HT receptor activation in mice.
Prog Neuropsychopharmacol Biol Psychiatry. Microarray and pathway analysis reveal distinct mechanisms underlying cannabinoid-mediated modulation of LPS-induced activation of BV-2 microglial cells. A cannabigerol quinone alleviates neuroinflammation in a chronic model of multiple sclerosis. Beneficial effect of the non-psychotropic plant cannabinoid cannabigerol on experimental inflammatory bowel disease.
Phytocannabinoids as novel therapeutic agents in CNS disorders. Inhibitory effect of cannabichromene, a major non-psychotropic cannabinoid extracted from Cannabis sativa, on inflammation-induced hypermotility in mice. Shinjyo N, Di Marzo V. Pharmacological evaluation of the natural constituent of Cannabis sativa, cannabichromene and its modulation by Delta 9 -tetrahydrocannabinol.
The plant cannabinoid Delta9-tetrahydrocannabivarin can decrease signs of inflammation and inflammatory pain in mice. Cannabinoid actions at TRPV channels: Acta Physiol Oxf ; 2: Novel effects of the cannabinoid inverse agonist AM on parameters related to metabolic syndrome in obese Zucker rats.
The CB 1 receptor antagonist, AM, improves recognition loss induced by naloxone in morphine withdrawal mice. Basic Clin Pharmacol Toxicol. Iran J Basic Med Sci.
Anti-tumor agent, attenuate catalepsy, immunosuppressive, inflammatory or anti-inflammatory agent depends upon used concentration of drug , antipsychotics [ - ].
Hepatic ischaemia, anti-inflammatory [ - ]. Anti-inflammatory [ ]. Analgesic, multiple sclerosis, neuroprotective [ ]. Anti-cancer agent, Analgesic, antiemetic, appetite stimulant. Analgesic, antiemetic, appetite stimulant, tumour growth inhibitor, multiple sclerosis [ ].
Anti-tumor,anti-inflammatory, antiemetic [ ]. Many people that use CBD to treat these diseases are usually more accepting of this method once they are fully educated on the it. It has little to no negative effects.
So many other methods that are used today to treat tumors such as: CBD downgrades ID-1 and inhibits breast cancer growth. The oil can also be a great treatment for the symptoms of the cancer which may include pains and aches. With its anti-tumor properties and pain relief, it makes the whole process for many women less painful and more bearable.
Many studies show that cannabinoid receptors are over-expressed in tumor cells of many cancers including breast cancer. This has led them to believe that the endocannabinoid system may be up-regulated in cancer in an innate biological effort to fight the disease. Cannabinoids have been proven to weaken angiogenesis increase in blood flow in localized areas caused by tumor cells and metastasis cancer reaching other organs.
In conclusion if God forbid, one gets breast cancer, the smartest thing to do would be investigate CBD oil and other products as it would be the least painful, least harmful, very effective and very easy method of treating this horrible disease. Comparing to conventional methods of treatment, this miracle oil does more good to the body while doing nothing negative and feeling most patients feeling better.
If no positive effects are noticed, try using different dosages or different ways of consuming CBD oil. All Medical Devices Medical Innovations.
How Does CBD Help with Breast Cancer?
While the benefits of CBD and medical marijuana are still being researched, many cancer patients have used the medicinal plant for symptom. A collection of published research articles and other educational resources about cancer and CBD (cannabidiol). Cannabis has been used medicinally for millennia, but has not been approved by the U.S. Food and Drug Administration to treat any medical.